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1.
Rev. Asoc. Odontol. Argent ; 103(3): 120-124, jul.-sept. 2015. ilus
Artigo em Espanhol | LILACS | ID: lil-768636

RESUMO

Objetivo: presentar la resolución quirúrgica de un caso clínico de recesiones gingivales tratadas con técnica de túnel y la evaluación de los resultados a los 180 días. Caso clínico: un paciente de sexo masculino, de 21 años de edad, concurrió a la consulta con recesiones de clase I de Miller, abfracciones e hipersensibilidad en las piezas 1.4 y 1.5. El procedimiento quirúrgico elegido fue la técnica de túnel. Se efectuó el seguimiento de la cicatrización y de la estabilidad de la cobertura radicular a los 180 días. Conclusión: el recubrimiento radicular de recesiones de clase I de Miller es altamente predecible, ya que permite anticipar una cobertura del 100 por ciento, siempre y cuando el enfoque quirúrgico esté acompañado del tratamiento de los factores etiológicos, así como támbién de la selección de una técnica adecuada


Assuntos
Humanos , Masculino , Adulto Jovem , Procedimentos Cirúrgicos Bucais/métodos , Raiz Dentária/lesões , Retração Gengival/cirurgia , Tecido Conjuntivo/transplante , Raspagem Dentária/métodos , Retalhos Cirúrgicos , Resultado do Tratamento
2.
Rev Argent Microbiol ; 45(3): 185-90, 2013.
Artigo em Espanhol | MEDLINE | ID: mdl-24165143

RESUMO

Sixty-four colistin-resistant Klebsiella pneumoniae isolates recovered from clinical specimens from 57 patients admitted to Hospital de Clinicas Jose de San Martin during the period 2010-2012 were studied to describe the microbiological and epidemiological characteristics and factors associated with the emergence of colistin-resistance. Fifty-four colistin-susceptible K. pneumoniae isolates from the same period were also included in the study. The genetic relatedness among the isolates was studied by a PCR assay. Fifty percent of the resistant isolates were KPC-2 producers, 45.3% were ESBL producers and 4.7% only showed resistance to aminopenicilins. All KPC-producers (resistant and susceptible to colistin) were genotipically indistinguishable except for one, whereas the presence of 7 clonal types, which were different from the ones identified in the colistin-susceptible isolates, were detected among ESBL producers. The previous use of colistin was the main factor associated with the acquisition of resistance, and in the case of non-KPC producers the stay in ICU was another significant factor observed. Colistin resistance emerged in our hospital in the year 2010, reaching 3% in nosocomial isolates and maintaining this rate in successive years, due to the selection of resistant subpopulations in the epidemic clonal type in KPC-producers and due to the dispersion of colistin-resistant clonal types in non-KPC producing-isolates.


Assuntos
Proteínas de Bactérias/biossíntese , Colistina/farmacologia , Klebsiella pneumoniae/efeitos dos fármacos , Klebsiella pneumoniae/enzimologia , beta-Lactamases/biossíntese , Proteínas de Bactérias/classificação , Farmacorresistência Bacteriana , Feminino , Humanos , Infecções por Klebsiella/epidemiologia , Masculino , beta-Lactamases/classificação
3.
Rev. argent. microbiol ; 45(3): 185-90, set. 2013.
Artigo em Espanhol | LILACS, BINACIS | ID: biblio-1171786

RESUMO

Sixty-four colistin-resistant Klebsiella pneumoniae isolates recovered from clinical specimens from 57 patients admitted to Hospital de Clinicas Jose de San Martin during the period 2010-2012 were studied to describe the microbiological and epidemiological characteristics and factors associated with the emergence of colistin-resistance. Fifty-four colistin-susceptible K. pneumoniae isolates from the same period were also included in the study. The genetic relatedness among the isolates was studied by a PCR assay. Fifty percent of the resistant isolates were KPC-2 producers, 45.3


were ESBL producers and 4.7


only showed resistance to aminopenicilins. All KPC-producers (resistant and susceptible to colistin) were genotipically indistinguishable except for one, whereas the presence of 7 clonal types, which were different from the ones identified in the colistin-susceptible isolates, were detected among ESBL producers. The previous use of colistin was the main factor associated with the acquisition of resistance, and in the case of non-KPC producers the stay in ICU was another significant factor observed. Colistin resistance emerged in our hospital in the year 2010, reaching 3


in nosocomial isolates and maintaining this rate in successive years, due to the selection of resistant subpopulations in the epidemic clonal type in KPC-producers and due to the dispersion of colistin-resistant clonal types in non-KPC producing-isolates.


Assuntos
Colistina/farmacologia , Klebsiella pneumoniae/efeitos dos fármacos , Klebsiella pneumoniae/enzimologia , Proteínas de Bactérias/biossíntese , beta-Lactamases/biossíntese , Farmacorresistência Bacteriana , Feminino , Humanos , Infecções por Klebsiella/epidemiologia , Masculino , Proteínas de Bactérias/classificação , beta-Lactamases/classificação
4.
Rev. Argent. Microbiol. ; 45(3): 185-90, 2013 Jul-Sep.
Artigo em Espanhol | BINACIS | ID: bin-132882

RESUMO

Sixty-four colistin-resistant Klebsiella pneumoniae isolates recovered from clinical specimens from 57 patients admitted to Hospital de Clinicas Jose de San Martin during the period 2010-2012 were studied to describe the microbiological and epidemiological characteristics and factors associated with the emergence of colistin-resistance. Fifty-four colistin-susceptible K. pneumoniae isolates from the same period were also included in the study. The genetic relatedness among the isolates was studied by a PCR assay. Fifty percent of the resistant isolates were KPC-2 producers, 45.3


were ESBL producers and 4.7


only showed resistance to aminopenicilins. All KPC-producers (resistant and susceptible to colistin) were genotipically indistinguishable except for one, whereas the presence of 7 clonal types, which were different from the ones identified in the colistin-susceptible isolates, were detected among ESBL producers. The previous use of colistin was the main factor associated with the acquisition of resistance, and in the case of non-KPC producers the stay in ICU was another significant factor observed. Colistin resistance emerged in our hospital in the year 2010, reaching 3


in nosocomial isolates and maintaining this rate in successive years, due to the selection of resistant subpopulations in the epidemic clonal type in KPC-producers and due to the dispersion of colistin-resistant clonal types in non-KPC producing-isolates.


Assuntos
Proteínas de Bactérias/biossíntese , Colistina/farmacologia , Klebsiella pneumoniae/efeitos dos fármacos , Klebsiella pneumoniae/enzimologia , beta-Lactamases/biossíntese , Proteínas de Bactérias/classificação , Farmacorresistência Bacteriana , Feminino , Humanos , Infecções por Klebsiella/epidemiologia , Masculino , beta-Lactamases/classificação
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